Please use this identifier to cite or link to this item: http://hdl.handle.net/1942/12807
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dc.contributor.advisorNAWROT, Tim-
dc.contributor.advisorSMEETS, Karen-
dc.contributor.authorJANSSEN, Bram-
dc.date.accessioned2011-11-25T09:06:57Z-
dc.date.available2011-11-25T09:06:57Z-
dc.date.issued2011-
dc.identifier.urihttp://hdl.handle.net/1942/12807-
dc.description.abstractAmbient particulate matter is of great concern to human health. In this cross-sectional study, we investigated the influence of PM10 exposure during pregnancy on the mtDNA-content, an established marker of mitochondrial damage and dysfunction. We hypothesized that mtDNA-content changed during pregnancy in response to PM10 exposure and may underlie susceptibility to mitochondrial dysfunction. Our findings indicate a potential window for susceptibility by trimester exposure that may adversely affect mitochondrial function and possibly foetal outcome. Placental mtDNA-content is associated with distance to major road, a surrogate for traffic-related air pollution.-
dc.format.mimetypeApplication/pdf-
dc.languagenl-
dc.language.isoen-
dc.publishertUL Diepenbeek-
dc.titleDecreased placental mitochondrial DNA-content in response to air pollution during in utero life-
dc.typeTheses and Dissertations-
local.bibliographicCitation.jcatT2-
dc.description.notesmaster in de biomedische wetenschappen-milieu en gezondheid-
local.type.specifiedMaster thesis-
dc.bibliographicCitation.oldjcatD2-
item.contributorJANSSEN, Bram-
item.fullcitationJANSSEN, Bram (2011) Decreased placental mitochondrial DNA-content in response to air pollution during in utero life.-
item.accessRightsOpen Access-
item.fulltextWith Fulltext-
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