Please use this identifier to cite or link to this item: http://hdl.handle.net/1942/14192
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dc.contributor.advisorKRAMER, Boris W.-
dc.contributor.authorDe Munter, Stephanie-
dc.date.accessioned2012-09-27T10:28:40Z-
dc.date.available2012-09-27T10:28:40Z-
dc.date.issued2012-
dc.identifier.urihttp://hdl.handle.net/1942/14192-
dc.description.abstractPerinatal hypoxic-ischemic encephalopathy (HIE) is a major cause of preterm brain injury. In the Western world, there has been a tremendous increase in preterm birth rates over the past 20 years and the incidence of perinatal HIE is estimated at two to six per 1000 live-born infants. Currently, few treatment options are available for preterms suffering from HIE. In this translational research project, we hypothesize that Mesenchymal Stem Cells (MSC) and Granulocyte-Colony Stimulating Factor (G-CSF) therapies can reduce hypoxic-ischemic related synapse loss, neurodegeneration, microglial proliferation and damage to pre-oligodendrocytes in a fetal sheep model, thereby limiting preterm HIE.-
dc.format.mimetypeApplication/pdf-
dc.languagenl-
dc.language.isoen-
dc.publishertUL Diepenbeek-
dc.titleCell therapy in neonatal hypoxic ischemic encephalopathy-
dc.typeTheses and Dissertations-
local.bibliographicCitation.jcatT2-
dc.description.notesmaster in de biomedische wetenschappen-klinische moleculaire wetenschappen-
local.type.specifiedMaster thesis-
dc.bibliographicCitation.oldjcatD2-
item.fulltextWith Fulltext-
item.accessRightsOpen Access-
item.fullcitationDe Munter, Stephanie (2012) Cell therapy in neonatal hypoxic ischemic encephalopathy.-
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